How to Manage Thyroid Conditions, Hypothyroidism, and Hashimoto’s: A Guide to Optimizing Your Health

In this episode of the School of Doza podcast, Nurse Doza discusses thyroid health, including hypothyroidism and Hashimoto's disease. He emphasizes the commonality of these conditions and provides advice for navigating them. Nurse Doza also explains the thyroid's role in the body and offers tips for improving thyroid function. The episode aims to provide listeners with knowledge and tools to optimize their health and transform their lives

 

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TIMESTAMPS:

00:00 CLASS IS IN SESSION

00:59 Thyroid Dysfunction & Hashimoto's

[00:03:41] Hashimoto's autoimmune disorder

[00:07:22] Hashimoto's and inflammation

[00:11:22] Hashimoto's and inflammation

[00:15:11] Hashimoto's and thyroid disorders

[00:17:40] Hashimoto's and TSH

[00:21:07] Thyroid Antibodies and Medication

[00:24:22] Hashimoto's and thyroid medication

[00:27:56] Stress and its effects

[00:31:31] Hashimoto's and the liver

[00:35:13] Liver health and inflammation

[00:38:26] Liver Love Supplement.

NOTES:

What: “Hashimoto’s thyroiditis is a type of autoimmune disease — your immune system doesn’t recognize your thyroid as your own and attacks it.

Hashimoto’s disease is common and affects about five people in 100 in the United States.”(1)

 

Who will develop ? “autoimmune diseases, like certain liver conditions, B12 deficiency, gluten sensitivity” (1)

Symptoms of Hashimoto disease include: (2)

• Weight gain
• Fatigue
• Hair loss
•  
• Low tolerance for cold temperatures
• Irregular menstrual periods
• Constipation
• Depression
• Joint pain

the patient may develop a non-tender, symmetrical, and painless goiter. As inflammation continues, thyroid follicles are damaged and can rupture. (3)

Heart disease: “Hypothyroidism lowers heart rate and cardiac output leading to increased risk of cardiovascular disease (Biondi et al. 2002; Biondi 2012), hypercholesterolemia, and hypertension (Klein and Ojamaa 2001; Toft and Boon 2000). Even subclinical hypothyroidism already presents a doubling of myocardial infarction and an elevated rate of atherosclerosis”(4)

Studies:https://my.clevelandclinic.org/health/diseases/17665-hashimotos-disease (1)

 

https://www.niddk.nih.gov/health-information/endocrine-diseases/hashimotos-disease(1)

https://medlineplus.gov/lab-tests/thyroid-antibodies/(2)

https://www.ncbi.nlm.nih.gov/books/NBK500006/ (3) 

Hs-crp hypothyroidism: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4244578/ (4)

The second thing you need to know about hashimotos/hypothyroidism TREATING TSH WONT MATTER________

 

“Hyperprolactinemia can be caused by hypothyroidism. Thyrotropin-releasing hormone (TRH) from the hypothalamus stimulates prolactin and TSH release. Prolactin release can suppress testosterone, LH, FSH, and GnRH release.” (1)

dx with the blood test TPO (thyroid perioxidase)______(2)

RT3: “T4 conversion to T3 and rT3: After its release from the thyroid gland, T4 is converted to T3, which is an active thyroid hormone, or to rT3, which is considered an inactive form. The rate and ratio of T4 conversion to either T3 or rT3 depend on the body’s metabolic needs.”(3)

Studies: https://www.ncbi.nlm.nih.gov/books/NBK500006/ (1)

https://medlineplus.gov/lab-tests/thyroid-antibodies/(2) 

https://www.verywellhealth.com/reverse-t3-thyroid-hormone-overview-3233184(3)

The third thing you need to know about hashimotos/hypothyroidism is medication might not fix it ______

 

Armour, NP (bioidentical) 

 

“How is Hashimoto’s disease treated?

If Hashimoto’s disease does progress to hypothyroidism, usual treatment is a synthetic (man-made) form of thyroid hormone called levothyroxine (Synthroid®, Tirosint®, Levoxyl®, Levothroid®, Unithroid®).”

“There is no special diet for Hashimoto’s disease”(1)

Studies: https://my.clevelandclinic.org/health/diseases/17665-hashimotos-disease(1)

The fourth thing you need to know about hashimotos/hypothyroidism is _your thyroid problem didnt start with the thyroid ______

 

HIGH DHEA-S: Infertility / acne / PCOS (1)

LOW DHEA-S: erectile dysfunction (1) 

 

Dopamine inhibits TSH synthesis and release (2), but raises TRH 

 

Epinephrine then cortisol in adrenals (3)

 

“After an external stimulus triggers the body’s stress response, the pituitary-adrenal axis and sympathetic division of the autonomic nervous system are activated.” (4)

 

“Functional deficiency due to impaired mechanisms of catecholamine release, reuptake, or receptor sensitivity has neurophysiologic effects involving dysregulation of mood and attention” (4)  “cocaine or amphetamines” 

 

“these findings indicate that the prolonged administration of pharmcological doses of DA significantly reduced serum TSH levels and thyroid hormone secretion in normal and criticall

 

ill patients, most likely by a direct inhibition of pituitary TSH with a secondary effect on thyroid gland secretion” (5) 

Studies: https://my.clevelandclinic.org/health/diagnostics/22148-dheas-test-dhea-sulfate-test (1)

 

HPA and thyroid https://www.ncbi.nlm.nih.gov/books/NBK278958/(2) 

 

Cortisol: https://www.ncbi.nlm.nih.gov/books/NBK538239/ (3) 

 

Catecholamines https://www.ncbi.nlm.nih.gov/books/NBK507716/ (4) 

 

Dopamine admi and thyroid suppression: https://pubmed.ncbi.nlm.nih.gov/7400302/ (5)

 

The fifth thing you need to know about hashimotos/hypothyroidism is _liver needs to be activated _______

Liver lowers TPO “there might exist common pathways in the pathogenesis of NAFLD and thyroid autoimmunity.” (1)

IL-6 inhibits TSH release (2)

IL-6 effects liver (3)

Studies: https://pubmed.ncbi.nlm.nih.gov/30016121/(1) 

https://www.ncbi.nlm.nih.gov/books/NBK278958/(2)

https://www.journal-of-hepatology.eu/article/S0168-8278(16)00083-0/fulltext(3)

 

Get 10% OFF MSW NUTRITION Supplements by use code NURSE DOZA

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